That Genereux post, what a long ass piece. As I'm writing this below, I anticipate my post will be long too.
Anyways, it's about auto-immune disorders. The basic premise is that it's genetic and/or environmental. It can't be genetic, that's clear enough. It must be environmental. That post suggests it's vitamin A poisoning. Maybe. I suggest that vitamin A poisoning is only possible if there's no fat coming in, or of course if the dose is high enough over a short enough time like 6 full bottles in a single dose of the stuff I used for my high-dose protocol. So, my protocol is 3 million IUs spread over 2 weeks taken with fat, lethal single dose is 56+ million IUs taken on its own (from mice experiments, so that number is a dose-per-bodyweight equivalent for humans).
The toxicity data is either from single lethal dose in mice or from very long-term low-dose like 10k IU daily over 6-10 years in humans. Correlation data is not reliable enough to make a determination, it suggests things, but it can't give us the power of prediction if we want to do something like I did for example. Modified vitamin A can't be directly extrapolated, it's not the same molecule, it has different effects. It's about the same as suggesting that sodium fluoride is the same as calcium fluoride.
The scale of time is important. Auto-immune disorders have not always been around. 100-200 years is not 2 million years. But 100-200 years certainly fits within our modern agricultural era, and the era of diseases of civilization. It's not just a new disease, it's a new environment. But that post also points out the very recent and dramatic rise of those diseases, so it's not just new, it's very new.
So, with auto-immune, the immune system is obviously involved. Well, vitamin A is essential for proper immune function, so the immune system is obviously involved here too.
So, I suggest vitamin A can become toxic if we don't eat enough fat. Not eating enough fat is kinda like drinking water in this era of low-fat high-carb standard American diet, everybody does it, some do it very well. Anyways, fat is required for absorption and metabolism of vitamin A. Not enough fat means we don't absorb it. Not enough fat means if we do absorb it, we don't metabolize it. If that's the case, it can become toxic because it lingers where it shouldn't. If this is what's happening, the simplest solution is to eat more fat, not just once or twice, but at every meal. Basically, we go low-carb.
Now what if we do eat enough fat, but then we supplement with vitamin A, but then it appears to be toxic with a bunch of symptoms? Well, it's not a matter of lingering vitamin A anymore - we eat enough fat, we metabolize it. It must be something else. So, we went low-carb, right? But it didn't fix everything.
Back to the immune system, cuz we're talking about auto-immune, and we're talking about vitamin A being essential for proper immune function. Now if we've determined it's not a problem of not eating enough fat, and it's not a problem of lingering vitamin A, then it can't be a problem with vitamin A, and it can't be a problem with proper immune function. It must be something else. In fact, we can't call it auto-immune anymore.
So, it must be a problem with the tissues and organs that are being attacked by a properly functioning immune system. So, what's the problem? Is it some genetic defect in those tissues and cells? Is it an infection we can't detect with the usual methods? I dunno what it is, but that's where the logic brings me.
It's just an alternative point of view, it's just how I see, not necessarily how it actually works. Take it with a grain of salt. But at least to me it makes sense to question the common view, or any view even my own.
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