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  #13   ^
Old Thu, Sep-16-21, 10:13
GRB5111's Avatar
GRB5111 GRB5111 is offline
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Posts: 4,036
 
Plan: Very LC, Higher Protein
Stats: 227/186/185 Male 6' 0"
BF:
Progress: 98%
Location: Herndon, VA
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Quote:
Originally Posted by teaser
If you read into the ketogenic diet for epilepsy, look for mechanisms of action. You'll find--direct effects of ketones. Effects of medium and short chain fatty acids that change when somebody is on a ketogenic diet. Effects of the ketogenic diet on essential fatty acid levels in the brain. Effects of the ketogenic diet on sodium and potassium channels. This is just top of my head, there's a lot more. Thing is, there's reasonable argument for lots of different routes by which it might be helpful. I don't think there has to be one over-arching explanation. Ketogenic metabolism is different enough from a more glucose-based metabolism--there's more than one point where things can go wrong with glucose metabolism, so it makes sense for there to be more than one mechanism of action when trying to sidestep much of it with a ketogenic diet. Appetite and bodyweight is also a complex system. Our body has to keep track of fat, carbohydrate and protein status and appetite(s), when a low carb--or even a low fat--diet works, it actually makes sense to me that there might be different mechanisms of action in different people.

Exactly and well stated. We've wasted a lot of time attempting to debate the "perfect human diet" when individual reactions to a way of eating may vary so much. People like Kevin Hall believe they've put an end to the proposed carbohydrate-insulin hypothesis when in reality, it can work so well for many. Insulin is hardly a killer, but managing it to achieve a healthy level is a good thing. Managing calories never worked for me in the traditional sense of controlling consumption. It worked when I ignored caloric measurement and simply ate the right ones for me.
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