From the graph, BG goes up to 120, then goes back down to 80. Before I got that nose infection, my BG stayed around 65 and never went much higher than that. I'm no athlete. Now imagine an athlete's BG. If an athlete is indeed exquisitely insulin sensitive, his BG will be much lower than mine was. If it's much lower, then even the tiniest rise in BG will cause a proportional rise in insulin, and this insulin will do its job of regulating glycolysis and lipolysis at every level.
Also, insulin sensitivity is like insulin level. When insulin resistance is high, it's like when insulin level is low, so more insulin is needed to do the same job. When insulin sensitivity is high, it's like when insulin level is high, so less insulin is needed to do the same job.
Then there's glycation - AGE's. HbA1c, one kind of AGE, is an indicator of average carb intake over the last several months. In other words, we suffer the consequences of carbohydrate poisoning even if we're exquisitely insulin sensitive. Athletes are not immune to the DOCs. In fact, since athletes are exquisitely insulin sensitive, it means their cells take up glucose more readily and quickly than the rest of us, and the damage is concentrated mostly inside their cells.
Then there's insulin sensitivity as a function of carbohydrate storage level. When cells have plenty of glucose, they become insulin resistant. This means when you feed an athlete who's insulin sensitive, their cells take in glucose more quickly, and become insulin resistant more quickly as well. Since insulin is used for amino acid uptake by cells, when we're insulin resistant our cells can't take up those amino acids as efficiently anymore, and we can't make as much maintenance and repair.
And BG level. When BG is low, cells will become insulin resistant to protect BG to save it for cells that absolutely need it. Now they're doing their natural job. But since BG is low, the need for insulin is much lower. If insulin is low, cells can go back to being insulin sensitive because there's isn't much insulin to push in the little glucose that's in the bloodstream. And when insulin is low, this is the signal for lipolysis so insulin sensitivity is less important. It's all relative.
Then there's cholesterol metabolism and its role in maintenance and repair. When we eat carbs, cholesterol metabolism is disrupted. Somehow I think that's a bad thing. Maintenance and repair suffers, we don't work as good anymore. An athlete's body works overtime, therefore accumulates extra wear and tear, therefore needs more maintenance and repair, not less.
Then there's the digestion factor. An athlete needs to be in top shape all the time. If what he eats causes digestive problems, his strength will be sapped. That's no good. I believe this super-starch isn't that good for digestion.
The point is that if we're just looking at the effect of super-starch on insulin, we're ignoring the big picture. Yet since athletes are exquisitely insulin sensitive, it means the tiniest changes in insulin level and sensitivity will produce much greater effects in athletes than in the rest of us. Indeed the tiniest changes in anything from hormones like testosterone to enzymes like HSL will produce much greater effects in them than in us. So when we're looking only at insulin level in athletes, we gotta change our perspective.
Basically it's the same thing with BP, heart rate, body fat, etc. All those things will be much better for an athlete than for the rest of us. Lower BP, HR, body fat, etc. BG should be lower as well. Mine was at 65 at rest and fasting. BP 90/60. HR 60. BF ~15%. All those things are regulated in part by insulin level and insulin sensitivity. Look at that graph again. That's not an athlete's graph, it's a regular people's graph.
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