Damn it, lost my whole post, t'was brilliant I say.

Ima recap the main points.

Episode 11 of ketohacking.

I wrote a blog post that predicts results of Jimmy's protein experiment, namely that BG will not rise.

He felt hypoglycemic, there's a simple explanation - too much insulin. But why is there too much insulin, and why is there still ketones? Liver insulin receptors shut down. Insulin can't get in, doesn't inhibit ketogenesis, yet acts on all other tissues cuz insulin circulates past the liver. Insulin doesn't get in the liver, liver doesn't degrade insulin, insulin stays too high.

He stayed hungry, in spite of too much insulin which should otherwise shut down hunger at the brain. Why? It's the dietary fat, not the ketones, not the insulin, just the dietary fat. Dietary fat activates the PPAR-x pathways in the liver. Therefore, dietary fat is a signaling molecule. This signal would then act on hunger and satiety hormones, ghrelin and leptin. That's where the hunger problem lies. It ain't the too much protein, it's the too little fat. This makes a lot of sense since eating too much fat is indeed quite difficult to do. The signaling from dietary fat comes straight from the gut, even as it is emulsified by bile, before it hits anything else.

I could try to re-write my brilliant post, but that'll do for now.

-edit- The thing about dietary fat signaling molecule, just came up with it in frustration about losing my brilliant post. Sorta brilliant in its own right if I say so myself.